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Old May 22nd, 2007, 07:46 PM
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Default How to fetch text after specific tag text

hi!

 I need to convert html file into structured xml. I have converted html into xhtml using jtidy.
I am facing problem with conversion of xhtml into xml file using xslt.

Below are xhtml file and output needed.

where after "<i>Background</i>" tag there is a plain text "this is background" i need to fetch this plain text (OUTPUT: <Background>this is background </Background>)
but problem is "this is background" is within <head> </head> tag. which also have plain text "this is the plain text" and can have any no of plain text.
In contrast, text after method can be found easily as it is witin tag <p> </p> (<p><i>Method </i> this is method</p>)


Small section of my xhml file's :

<head>
this is the plain text.
<title>this is title</title>
this is all plain text under head

<i>Background</i> this is background
<p><i>Method</i>this is method </p>
</head>

 OUTPUT XML FILE :

<Title>this is title</Title>
<Background>this is background</Background>
<Method>this is method</Method>

 Is there any way to insert tag after Background and before Method so that i can get that specific text which is between background and method.

Thanks in advance.
komal

 
Old May 23rd, 2007, 01:32 AM
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Well you need to be more specific about the structure, are the terms background and method always there or do you need a more general solution?
For the input you have shown, which is atrocious XHTML, you can use:
Code:
<xsl:stylesheet version="2.0"
  xmlns:xsl="http://www.w3.org/1999/XSL/Transform">
  <xsl:template match="/">
    <data>
      <title><xsl:value-of select="*/title"/></title>
      <xsl:apply-templates select="//i"/>
    </data>
  </xsl:template>

  <xsl:template match="i">
    <xsl:element name="{.}">
      <xsl:value-of select="following-sibling::text()[1]"/>
    </xsl:element>
  </xsl:template>
</xsl:stylesheet>
If it really is XHTML then you'll have to add the xhtml namespace mappings, see the first post in this forum for guidance if you're not sure how to do this.

--

Joe (Microsoft MVP - XML)
 
Old May 23rd, 2007, 03:42 AM
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If $b is the element <i>Background</i> then the text that immediately follows is $b/following-sibling::text()[1]

Michael Kay
http://www.saxonica.com/
Author, XSLT Programmer's Reference and XPath 2.0 Programmer's Reference
 
Old May 23rd, 2007, 10:15 AM
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Thanks, but i understand the apporch of choosing text()[1] but in my actual file there could be any no of line of plain text above that
i need to find dynamically position of text .. it could be 1 or n.. i don't know.
for example

my code need to work for general case.
<head>
bla .......................bla.....
this is header like ..........

<i>Background</i> this is backgroud
<p><i>Method</i>this is method</p>


this is end of header ... bla ... bla

</head>

In actual file java script is also present. I am learning with small file right now as i need to built parser for my school project work. I havn't tried with my actual files.
i have currently downloaded all html file and converted into xhtml. for example html file i hv to parse at :
http://content.onlinejacc.org/cgi/co...bstract/41/1/1

Once again thank you very much for suggestions.

 
Old May 23rd, 2007, 10:40 AM
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hello!
        I got my point,i ask in last post. I am really sorry asking you guys again.but finding something unsual problem while working with my actual xhtml to xml file. i am using
javax.xml.transform.* to parse my xhtml file using xslt. When i am trying to fetch very simple node of xhtml file.
for examle: <title> </title>
it's been getting me plain text output of whole file. i don't understand why it gives me the text of whole file while i am trying to get only text of <title></title>.

pl. clear this basic doubt of mine.

:PARTIAL OUTPUT OF XHTML:

<!DOCTYPE html PUBLIC "-//W3C//DTD XHTML 1.0 Transitional//EN"
    "http://www.w3.org/TR/xhtml1/DTD/xhtml1-transitional.dtd">

<html
 xmlns="http://www.w3.org/1999/xhtml">
<head>
<meta
 name="generator"
 content="HTML Tidy, see www.w3.org" />
<title>

    Association between hyperglycemia and the no-reflow phenomenon inpatients with acute myocardial infarction -- Iwakura et al. 41 (1): 1 -- Journal of the American College of Cardiology</title>


 thanks
Komal

 
Old May 23rd, 2007, 10:40 AM
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Well can you show an XHTML file for which the above doesn't work? Please be precise and eliminate unnecessary content beforehand.

--

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Old May 23rd, 2007, 10:49 AM
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Look at the first answer I gave regarding namespaces.

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Old May 23rd, 2007, 11:01 AM
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here is the actual xhtml file and problem is it parse the whole file while i am trying only for title node.


<!DOCTYPE html PUBLIC "-//W3C//DTD XHTML 1.0 Transitional//EN"
    "http://www.w3.org/TR/xhtml1/DTD/xhtml1-transitional.dtd">

<html
 xmlns="http://www.w3.org/1999/xhtml">
<head>
<meta
 name="generator"
 content="HTML Tidy, see www.w3.org" />
<title>Association between hyperglycemia and the no-reflow
phenomenon inpatients with acute myocardial infarction -- Iwakura
et al. 41 (1): 1 -- Journal of the American College of
Cardiology</title>
<meta
 http-equiv="Content-Type"
 content="text/html; charset=windows-1252" />

<script
 type="text/javascript"
 language="JavaScript">
    <!--
    if ( top != self )
    {
        top.location.href = unescape(window.location.pathname);
    }
    //-->

</script>

<style
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    PADDING-RIGHT: 0px; PADDING-LEFT: 10px; PADDING-BOTTOM: 5px; PADDING-TOP: 0px
}
TABLE.content_box_outer_table_in_sidebar_frame {
    PADDING-RIGHT: 10px; PADDING-LEFT: 10px; PADDING-BOTTOM: 0px; PADDING-TOP: 10px
}
TABLE.content_box_inner_table {
    PADDING-RIGHT: 0px; PADDING-LEFT: 0px; FONT-SIZE: 65%; PADDING-BOTTOM: 5px; PADDING-TOP: 0px; FONT-FAMILY: Verdana,Arial,Helvetica,sans-serif; BACKGROUND-COLOR: #f0f0f0
}
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}
TD.content_box_title_highlight {
    PADDING-RIGHT: 1px; PADDING-LEFT: 1px; FONT-WEIGHT: bold; FONT-SIZE: 120%; PADDING-BOTTOM: 1px; VERTICAL-ALIGN: middle; COLOR: #ffffff; PADDING-TOP: 1px; FONT-STYLE: italic; FONT-FAMILY: "Trebuchet MS",Verdana,Arial,Helvetica,sans-serif; BACKGROUND-COLOR: #003399; TEXT-ALIGN: center
}
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}
TD.content_box_space_between_sections {
    PADDING-TOP: 2px
}
TD.content_box_arrow {
    PADDING-RIGHT: 0px; PADDING-LEFT: 5px; PADDING-BOTTOM: 3px; PADDING-TOP: 0px; TEXT-ALIGN: right
}
TD.content_box_item {
    PADDING-RIGHT: 5px; PADDING-LEFT: 3px; PADDING-BOTTOM: 3px; VERTICAL-ALIGN: top; PADDING-TOP: 0px; TEXT-ALIGN: left
}
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}
FONT.openaccess {
    PADDING-RIGHT: 3px; PADDING-LEFT: 3px; FONT-WEIGHT: bold; BACKGROUND-COLOR: #ffffff
}
FONT.openaccess_open {
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}
FONT.openaccess_access {
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}
FONT.openaccess_oa {

}
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}
DIV.referrer-based-search-line {
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}
DIV.referrer-based-search-line .search-terms {
    FONT-WEIGHT: bold
}
DIV.referrer-based-search-line .collection-name {
    FONT-STYLE: italic
}

@media Print, Screen, Handheld
{
.notonscreen {
    DISPLAY: none
}

}
</style>

<script
 type="text/javascript"
 language="javascript">
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 content="12570936"
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 content="Iwakura, Katsuomi"
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 valign="top"
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<table>
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<tr>
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<td
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</td>
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<td
 valign="top">J Am Coll Cardiol, 2003; 41:1-7<br />
<a
 href="http://content.onlinejacc.org/misc/terms.dtl">&copy; 2003 by
the American College of Cardiology Foundation</a><br />
</td>
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<h4>CLINICAL STUDY: MYOCARDIAL INFARCTION AND ACUTE CORONARY
SYNDROME</h4>

<h2>Association between hyperglycemia and the no-reflow phenomenon
inpatients with acute myocardial infarction</h2>

<nobr>Katsuomi Iwakura, MD<sup>*</sup></nobr>, <nobr>Hiroshi Ito,
MD, FACC<sup>*</sup><sup>,*</sup></nobr>, <nobr>Masashi Ikushima,
MD<sup>*</sup></nobr>, <nobr>Shigeo Kawano, MD<sup>*</sup></nobr>,
<nobr>Atsushi Okamura, MD<sup>*</sup></nobr>, <nobr>Katsuaki Asano,
MD<sup>*</sup></nobr>, <nobr>Tadashi Kuroda, MD<sup>*</sup></nobr>,
<nobr>Koji Tanaka, MD<sup>*</sup></nobr>, <nobr>Tohru Masuyama,
MD<sup><img
 alt="{dagger}"
 src="journalacc_files/dagger.gif"
 border="0" /></sup></nobr>, <nobr>Masatsugu Hori, MD<sup><img
 alt="{dagger}"
 src="journalacc_files/dagger.gif"
 border="0" /></sup></nobr> and <nobr>Kenshi Fujii,
MD<sup>*</sup></nobr>

<p><sup>*</sup> Division of Cardiology, Sakurabashi Watanabe
Hospital, Osaka University, Osaka, Japan<br />
<sup><img
 alt="{dagger}"
 src="journalacc_files/dagger.gif"
 border="0" /></sup> Department of Internal Medicine and
Therapeutics, Graduate School of Medicine, Osaka University, Osaka,
Japan</p>

<p>Manuscript received August 13, 2002; revised manuscript received
September 12, 2002, accepted September 20, 2002.</p>

<p><sup>*</sup> <b>Reprint requests and correspondence:</b> Dr.
Hiroshi Ito, Division of Cardiology, Sakurabashi Watanabe Hospital,
2-4-32 Umeda, Kita-ku, Osaka 530-0001, Japan. <sup></sup><br />
<span
 id="em0">itomd{at}osk4.3web.ne.jp</span>

<script
 type="text/javascript">
<!--
 var u = "itomd", d = "osk4.3web.ne.jp"; document.getElementById("em0").innerHTML = '<a href="mailto:' + u + '@' + d + '">' + u + '@' + d + '<\/a>'//-->
</script>
</p>

<p>OBJECTIVES: We investigated the association between
hyperglycemia and the <sup></sup>no-reflow phenomenon in patients
with acute myocardial infarction <sup></sup>(AMI). <sup></sup></p>

<p>BACKGROUND: Hyperglycemia is associated with increased risks of
heart failure, <sup></sup>cardiogenic shock, and death after AMI,
but its underlying mechanism <sup></sup>remains unknown.
<sup></sup></p>

<p>METHODS: A total of 146 consecutive patients with a first AMI
were studied <sup></sup>by intracoronary myocardial contrast
echocardiography (MCE) <sup></sup>after successful reperfusion
within 24 h after symptom onset. <sup></sup>Two-dimensional
echocardiography was recorded on day 1 and three <sup></sup>months
later to determine the change in the wall motion score
<sup></sup>(<img
 alt="{Delta}"
 src="journalacc_files/Delta.gif"
 border="0" />WMS; sum of 16 segmental scores; dyskinesia = 4 to
normokinesia <sup></sup>= 0). <sup></sup></p>

<p>RESULTS: The no-reflow phenomenon was found on MCE in 49 (33.6%)
of 146 <sup></sup>patients; their glucose level on hospital
admission was significantly <sup></sup>higher than that of patients
who did not exhibit this phenomenon <sup></sup>(209 &plusmn; 79 vs.
159 &plusmn; 56 mg/dl; p &lt; 0.0001). <sup></sup>There was no
difference in glycosylated hemoglobin or in the
<sup></sup>incidence of diabetes mellitus between the two subsets.
The <sup></sup>no-reflow phenomenon was more often observed in the
75 patients <sup></sup>with hyperglycemia (<img
 alt="&ge;"
 src="journalacc_files/ge.gif"
 border="0" />160 mg/dl) than in those without hyperglycemia
<sup></sup>(52.0% vs. 14.1%; p &lt; 0.0001). Patients with
hyperglycemia <sup></sup>had a higher peak creatine kinase level
(2,497 &plusmn; 1,603 <sup></sup>vs. 1,804 &plusmn; 1,300 IU/l; p =
0.005) and a lower <img
 alt="{Delta}"
 src="journalacc_files/Delta.gif"
 border="0" />WMS (3.7 <sup></sup>&plusmn; 4.8 vs. 5.7 &plusmn;
4.3; p = 0.01) than did those <sup></sup>without hyperglycemia. The
blood glucose level was an independent <sup></sup>prognostic factor
for no reflow, along with age, gender, absence <sup></sup>of
pre-infarction angina, complete occlusion of the culprit
<sup></sup>lesion, and anterior AMI. <sup></sup></p>

<p>CONCLUSIONS: Hyperglycemia might be associated with impaired
microvascular <sup></sup>function after AMI, resulting in a larger
infarct size and worse <sup></sup>functional recovery.
<sup></sup></p>
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<h4>CLINICAL STUDY: MYOCARDIAL INFARCTION AND ACUTE CORONARY
SYNDROME</h4>

<h2>Association between hyperglycemia and the no-reflow phenomenon
inpatients with acute myocardial infarction</h2>

<nobr>Katsuomi Iwakura, MD<sup>*</sup></nobr>, <nobr>Hiroshi Ito,
MD, FACC<sup>*</sup><sup>,*</sup></nobr>, <nobr>Masashi Ikushima,
MD<sup>*</sup></nobr>, <nobr>Shigeo Kawano, MD<sup>*</sup></nobr>,
<nobr>Atsushi Okamura, MD<sup>*</sup></nobr>, <nobr>Katsuaki Asano,
MD<sup>*</sup></nobr>, <nobr>Tadashi Kuroda, MD<sup>*</sup></nobr>,
<nobr>Koji Tanaka, MD<sup>*</sup></nobr>, <nobr>Tohru Masuyama,
MD<sup><img
 alt="{dagger}"
 src="journalacc_files/dagger.gif"
 border="0" /></sup></nobr>, <nobr>Masatsugu Hori, MD<sup><img
 alt="{dagger}"
 src="journalacc_files/dagger.gif"
 border="0" /></sup></nobr> and <nobr>Kenshi Fujii,
MD<sup>*</sup></nobr>

<p><sup>*</sup> Division of Cardiology, Sakurabashi Watanabe
Hospital, Osaka University, Osaka, Japan<br />
<sup><img
 alt="{dagger}"
 src="journalacc_files/dagger.gif"
 border="0" /></sup> Department of Internal Medicine and
Therapeutics, Graduate School of Medicine, Osaka University, Osaka,
Japan</p>

<p>Manuscript received August 13, 2002; revised manuscript received
September 12, 2002, accepted September 20, 2002.</p>

<p><sup>*</sup> <b>Reprint requests and correspondence:</b> Dr.
Hiroshi Ito, Division of Cardiology, Sakurabashi Watanabe Hospital,
2-4-32 Umeda, Kita-ku, Osaka 530-0001, Japan. <sup></sup><br />
<span
 id="em0">itomd{at}osk4.3web.ne.jp</span>

<script
 type="text/javascript">
<!--
 var u = "itomd", d = "osk4.3web.ne.jp"; document.getElementById("em0").innerHTML = '<a href="mailto:' + u + '@' + d + '">' + u + '@' + d + '<\/a>'//-->
</script>
</p>

<p>OBJECTIVES: We investigated the association between
hyperglycemia and the <sup></sup>no-reflow phenomenon in patients
with acute myocardial infarction <sup></sup>(AMI). <sup></sup></p>

<p>BACKGROUND: Hyperglycemia is associated with increased risks of
heart failure, <sup></sup>cardiogenic shock, and death after AMI,
but its underlying mechanism <sup></sup>remains unknown.
<sup></sup></p>

<p>METHODS: A total of 146 consecutive patients with a first AMI
were studied <sup></sup>by intracoronary myocardial contrast
echocardiography (MCE) <sup></sup>after successful reperfusion
within 24 h after symptom onset. <sup></sup>Two-dimensional
echocardiography was recorded on day 1 and three <sup></sup>months
later to determine the change in the wall motion score
<sup></sup>(<img
 alt="{Delta}"
 src="journalacc_files/Delta.gif"
 border="0" />WMS; sum of 16 segmental scores; dyskinesia = 4 to
normokinesia <sup></sup>= 0). <sup></sup></p>

<p>RESULTS: The no-reflow phenomenon was found on MCE in 49 (33.6%)
of 146 <sup></sup>patients; their glucose level on hospital
admission was significantly <sup></sup>higher than that of patients
who did not exhibit this phenomenon <sup></sup>(209 &plusmn; 79 vs.
159 &plusmn; 56 mg/dl; p &lt; 0.0001). <sup></sup>There was no
difference in glycosylated hemoglobin or in the
<sup></sup>incidence of diabetes mellitus between the two subsets.
The <sup></sup>no-reflow phenomenon was more often observed in the
75 patients <sup></sup>with hyperglycemia (<img
 alt="&ge;"
 src="journalacc_files/ge.gif"
 border="0" />160 mg/dl) than in those without hyperglycemia
<sup></sup>(52.0% vs. 14.1%; p &lt; 0.0001). Patients with
hyperglycemia <sup></sup>had a higher peak creatine kinase level
(2,497 &plusmn; 1,603 <sup></sup>vs. 1,804 &plusmn; 1,300 IU/l; p =
0.005) and a lower <img
 alt="{Delta}"
 src="journalacc_files/Delta.gif"
 border="0" />WMS (3.7 <sup></sup>&plusmn; 4.8 vs. 5.7 &plusmn;
4.3; p = 0.01) than did those <sup></sup>without hyperglycemia. The
blood glucose level was an independent <sup></sup>prognostic factor
for no reflow, along with age, gender, absence <sup></sup>of
pre-infarction angina, complete occlusion of the culprit
<sup></sup>lesion, and anterior AMI. <sup></sup></p>

<p>CONCLUSIONS: Hyperglycemia might be associated with impaired
microvascular <sup></sup>function after AMI, resulting in a larger
infarct size and worse <sup></sup>functional recovery.
<sup></sup></p>
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Old May 23rd, 2007, 12:08 PM
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